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Nandrolone (Deca Durabolin) Nandrolone is one of the most commonly used steroids for muscle growth. In an effort to enhance the effects of this steroid, some authors have considered its use alongside other GH stimulators. The purpose of this paper is to evaluate the effects of Nandrolone on muscular growth, and to examine the relationship between Nandrolone and GH levels in the GH-deficient state. In this context, a GH-deficient state is defined as an absence of GH release in the circulation or without GH production via an oral GH or an injectable GH analogue, or a state of low GH secretion that is associated with GH deficiency. Results from an in vivo, in vitro study assessing GH secretion rate and GH responses after oral administration of Nandrolone (2-8 mg) confirmed that Nandrolone stimulates GH release, with the increase in GH levels being largest with highest doses and greatest time to peak. Results from an in vitro study demonstrating that Nandrolone is an inhibiting factor on GH release from different tissues provide insights into the mechanism by which Nandrolone exerts its inhibitory effects on GH release. The ability of Nandrolone to increase plasma GH concentrations has been demonstrated in vitro, and Nandrolone administration leads to a more pronounced GH effect in humans than that seen with oral GH treatment. It seems that the inhibitory effect of Nandrolone on GH release is due, in part, to inhibition of GH's reuptake. In humans, Nandrolone administration inhibits GH secretion while providing no significant increase in GH concentrations. Thus, with the use of Nandrolone in humans for the study of the GH-suppressant action, it will be important to determine whether Nandrolone enhances the effect on GH secretion, at least in part, by inhibiting its reuptake. Thus, in addition to determining the effect of Nandrolone on GH release from muscles, it will likely be valuable to determine the effect of Nandrolone on GH levels and how they are affected by Nandrolone administration in the GH deficient state. Footnotes Disclosures: The authors disclosed no conflicts of interest to report. Correspondence: Nandrolone, National Institute on Drug Abuse, P.O. Box 78532, Rockville, MD 20852-732; e-mail: email@example.com . Related Article: